Kelvin J. A. Davies
TitleCited byYear
Mitochondrial free radical generation, oxidative stress, and aging
E Cadenas, KJA Davies
Free Radical Biology and Medicine 29 (3-4), 222-230, 2000
30842000
Free radicals and tissue damage produced by exercise
KJA Davies, AT Quintanilha, GA Brooks, L Packer
Biochemical and biophysical research communications 107 (4), 1198-1205, 1982
22051982
Protein damage and degradation by oxygen radicals. I. general aspects.
KJ Davies
Journal of Biological Chemistry 262 (20), 9895-9901, 1987
21831987
Oxidative stress: the paradox of aerobic life
KJA Davies
Biochemical Society Symposia 61, 1-31, 1995
14721995
Measuring reactive oxygen and nitrogen species with fluorescent probes: challenges and limitations
B Kalyanaraman, V Darley-Usmar, KJA Davies, PA Dennery, HJ Forman, ...
Free Radical Biology and Medicine 52 (1), 1-6, 2012
10712012
Degradation of oxidized proteins in mammalian cells.
T Grune, T Reinheckel, KJ Davies
The FASEB Journal 11 (7), 526-534, 1997
9201997
Oxidative stress, antioxidant defenses, and damage removal, repair, and replacement systems
KJA Davies
IUBMB life 50 (4‐5), 279-289, 2000
9082000
Protein damage and degradation by oxygen radicals. II. Modification of amino acids.
KJ Davies, ME Delsignore, SW Lin
Journal of Biological Chemistry 262 (20), 9902-9907, 1987
8861987
Degradation of oxidized proteins by the 20S proteasome
KJA Davies
Biochimie 83 (3-4), 301-310, 2001
8332001
Calcium and oxidative stress: from cell signaling to cell death
G Ermak, KJA Davies
Molecular immunology 38 (10), 713-721, 2002
7742002
The oxidative inactivation of mitochondrial electron transport chain components and ATPase.
Y Zhang, O Marcillat, C Giulivi, L Ernster, KJ Davies
Journal of Biological Chemistry 265 (27), 16330-16336, 1990
7681990
Protein damage and degradation by oxygen radicals. III. Modification of secondary and tertiary structure.
KJ Davies, ME Delsignore
Journal of Biological Chemistry 262 (20), 9908-9913, 1987
6501987
Decreased proteolysis caused by protein aggregates, inclusion bodies, plaques, lipofuscin, ceroid, and ‘aggresomes’ during oxidative stress, aging, and disease
T Grune, T Jung, K Merker, KJA Davies
The international journal of biochemistry & cell biology 36 (12), 2519-2530, 2004
6112004
Redox cycling of anthracyclines by cardiac mitochondria. I. Anthracycline radical formation by NADH dehydrogenase.
KJ Davies, JH Doroshow
Journal of Biological Chemistry 261 (7), 3060-3067, 1986
5841986
Uric acid-iron ion complexes. A new aspect of the antioxidant functions of uric acid
KJA Davies, A Sevanian, SF Muakkassah-Kelly, P Hochstein
Biochemical Journal 235 (3), 747-754, 1986
5771986
Redox cycling of anthracyclines by cardiac mitochondria. II. Formation of superoxide anion, hydrogen peroxide, and hydroxyl radical.
JH Doroshow, KJ Davies
Journal of Biological Chemistry 261 (7), 3068-3074, 1986
5401986
The broad spectrum of responses to oxidants in proliferating cells: a new paradigm for oxidative stress
KJA Davies
IUBMB life 48 (1), 41-47, 1999
5241999
Protein damage and degradation by oxygen radicals. IV. Degradation of denatured protein.
KJ Davies, SW Lin, RE Pacifici
Journal of Biological Chemistry 262 (20), 9914-9920, 1987
5131987
Oxygen radicals stimulate intracellular proteolysis and lipid peroxidation by independent mechanisms in erythrocytes.
KJ Davies, AL Goldberg
Journal of Biological Chemistry 262 (17), 8220-8226, 1987
5001987
Lon protease preferentially degrades oxidized mitochondrial aconitase by an ATP-stimulated mechanism
DA Bota, KJA Davies
Nature cell biology 4 (9), 674, 2002
4972002
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